Ok so I have read that as you get older you produce less None. Additionally it has been discussed here that Asians produce less None as well. So for an Asian guy like myself in their 30’s how much None should I use to produce the appropriate effects. For example most people tend to use 2-3 drops of A314 or with Ammo maybe 2-3 sprays. Should I increase this amount to 4-5? Same with something like TUTH. Do I go for 2-3 sprays? Cheers dudes.
Hmm, it’s hard to pinpoint how much None you should use. It depends on person to person. Some asians OD quickly with even one drop, Gegogi (member of these forums) can practically shower in it and never OD. So you should experiment a little and find an even balance. Have fun with your pheros btw.
so I was doing research to try and answer this question when I came across this abstract on testosterone.
Chronic endogenous testosterone exposure adversely affects proinflammatory and proapoptotic signaling after ischemia/reperfusion; however, it remains unknown whether a single acute testosterone exposure is equally detrimental. We hypothesized that acute exogenous testosterone infusion before ischemia would worsen myocardial functional recovery, increase the activation of MAPKs and caspase-3, and increase myocardial proinflammatory cytokine production. To study this, isolated-perfused rat hearts (Langendorff) from adult females and castrated males were subjected to 25-min ischemia and 40-min reperfusion with and without acute testosterone infusion (17[beta]-hydroxy-4-androstenone, 10 ng*[ml.sup.-1]*[min.sup.-1]) before ischemia. Myocardial contractile function was continuously recorded. After ischemia/reperfusion, hearts were assessed for levels of testosterone (ELISA), expression of proinflammatory cytokines (ELISA), and activation of MAPKs and caspase-3 (Western blot analysis). Data were analyzed with two-way ANOVA or Student’s t-test; P < 0.05 was statistically significant. All indices of postischemic functional recovery were decreased with acute exogenous testosterone compared with the untreated groups. Acute testosterone infusion increased activation of MAPKs and caspase-3 following ischemia/reperfusion. However, there were no significant differences in the myocardial proinflammatory cytokine production after brief testosterone infusion. A single acute exposure to exogenous testosterone before ischemia worsens myocardial functional recovery and increases activation of MAPKs and caspase-3. These findings confirm the deleterious effects of testosterone on myocardium, elucidate the nongenomic mechanistic pathways of testosterone, and may have important clinical implications for patients who have acute exposure to exogenous testosterone.
Words to know for this article:
myocardium - muscle tissue in the heart.
ischemia - decrease in blood supply usually cause by something blocking/hindering the flow of blood in the vessels.
exogenous - something thats produced outside of the organism. All pheromones are exogenous hormones just FYI.
This comes from a peer reviewed journal article from The American Journal of Physiology.
My, dem dars ah mighty heap o’ words. Made me horny…
well… its not a good thing.
Basically they found that testosterone has a negative effect on ischemia recovery when exposed to high levels of testosterone. I guess it doesn’t really matter if you don’t smoke, have good cholesterol levels and good blood pressure or have had a heart attack/stroke.
Zeph..you’re showing your asian. 
LOLOL. Oh man. GG NO RE? NR15? GL HF?
I’m assuming you play SC though. I couldn’t help it, it got me curious if certain people had different none levels or what not. It’s just sad that there’s very little on the subject matter. The scientific community is just starting to research pheromones.
JVK’s tome on pheromones, Scent of Eros, addresses the issue of pheromone production among different racial groups, albeit in layman’s terms and is an interesting read to boot. Judging from all the abstracts from the last 20 years it’s more than “just starting.” Merely has a long ways to go.
mmm well from the doctors I’ve been talking to (older doctors) they all seem to have the stance that pheromones are meant for animals and that humans don’t really use them. A lot of research and studies I’ve been looking up are barely willing to admit that they exist. There are a few studies that prove that they do but even then, they are reluctant to say that there is proof that pheromones affect humans too.
When I get back from class I’ll put up a study or two. Just FYI I use the UC system through the UCI library to do my research.
I suppose “just starting” IS a bad way to describe it. It’s more like they’re actually taking it seriously now. Kinda like how it took a while for the field of psychology to be admitted as a science.
I guess its kinda like Mendel and his paper on genetic alleles. Undiscovered for decades after his death.
Yea, of course I played SC. For our gen, it seems almost like you’re not korean if you haven’t played SC..I haven’t played it for a while though..waiting on the sequel.
Clearly the actual studying of ‘pheromones’ has not just started. I think what you meant was that it seems that pheromones are starting to become more recognized as a legitimate topic for research in the scientific community. But, I’m sure some of the members on this forum would disagree. Some have been using pheromones 2x, maybe 3x longer than I’ve even known about pheromones. They might assert that relative to how it is now, pheromones have been out of the closet for some time.
Actually, Phya, that was exactly what I was trying to say. Its becoming a more legitimate area of study.
Oh, good. Glad I hit it on the head. 